The medical term for baldness. Alopecia can be temporary or permanent. It occurs due to heredity, diseases, functional disorders or traumas.
Can be of hereditary, psychological or immunological origin. In case all hair of scalp and face are lost it is called Alopecia Totalis and when hairs are lost from all the body it is called Alopecia Universalis.
Hair loss is observed 1 to 3 weeks after the exposure to one of the previous factors. In most cases, hair regrows when the cause of Anagen Effluvium ceases.
It also affects women though with differences compared to men (FEMALE PATTERN HAIR LOSS).
It occurs due to the long-lasting impact of androgens to hair follicles with hereditary predisposition to hair loss.
The prime representative of androgens is testosterone.
Long-lasting impact of androgens on susceptible hair follicles brings about their gradual miniaturization and leads to Androgenetic Alopecia, the most common type of hair loss in men.
Hair follicles start shrinking, hair growth stops and hairs are being prepared to fall out. Approximately 1 to 2% of hair lie in the Catagen Phase.
Derivative of testosterone.
Testosterone, with the help of the enzyme 5-a reductase is converted into DHT, which due to its presence on susceptible hair follicles is responsible for hair miniaturization (gradual shrinking of hair follicles that leads to gradual shrinking of hair).
Hair loss is the final stage of hair miniaturization.
Transplanted hair follicles preserve their genetic resistance to androgens even in the thinning area and continue to grow normally throughout a life time.
This principle has been the basis for the foundation and development of modern hair transplantation.
It states that hair follicles from the donor area preserve their genetic resistance to androgens and continue to grow throughout a lifetime.
The extraction of hair follicles and their reimplantation to the area of hair loss has no impact on their function and they continue to grow normally throughout a life time.
Hair follicle cloning is the experimental technique described from the following steps:
- Extraction of cells from hair follicles.
- Multiplication of extracted cells in vitro.
- Reimplantation of the new cells on the scalp in order to produce new hair follicles.
Researchers made trials on wounded mice: they took skin tissue of wounded mice and by using wnt protein they observed that cells of the epidermis present the properties of stem cells and regenerate hair follicles.
New hair follicles can produce hair shafts which grow normally through all stages of the hair growth cycle.
This outcome has created many hopes for hair loss treatment in man, as researchers have decoded the genes involved in the production of new hair.
Hair grows in naturally occurring group of 1-4 hairs, called Hair Follicles or Follicular Units.
Hair follicles are complete biological structures that comprise nerves, blood vessels, sebaceous glands and the small arrectorpili: this muscle lies at the base of hair follicle and in any excitement due to cold or fear, forces shaft to rise.
Hair follicles are surrounded by protective, connective collagen tissue and lie everywhere in the body except lips, palms and soles.
All hair follicles have a predefined genetic definition. Those consisting of one hair are allocated along the frontal hairline and as we move towards the back of the scalp, hair follicles constitute groups of 2, 3 or 4 hairs.
The acronym for the International Society of Hair Restoration Surgery, the non-profit voluntary organization of surgeon specialists for the advance of hair restoration surgery.
The graphic representation of Female Pattern Baldness degrees.
Keratin is a group of proteins that contain sulphur and is being produced in the keratogen zone of the root.
It contributes to the strength and resilience of the hair shaft and to nail configuration as well.
Hair consists of proteins (65% – 95%), lipids (1% – 9%), trace elements, polysaccharides and water. Keratin also lies in the skin.
Hair transplant sessions of more than 2.500 follicular units.
The graphic representation of Androgenetic Alopecia degrees.
Trichotillomania is a form of self-enforced scarring alopecia, in which the affected person develops the need to pull and uproot his own hair, usually by rolling his hair around his finger.
Telogen Effluvium is a form of alopecia characterized by diffuse hair shedding.
There are 3 types of Telogen Effluvium: acute, sub acute and chronic.
The interruption of the normal hair growth cycle can occur as a result of metabolic stress (chemotherapy, pregnancy, puberty, surgery, severe stress and chronic illnesses), emotional trauma or systematic attack.
Hair loss appears 3 months after the experience of the intense incident and it may involve even 50% of hair in rare cases.
The third stage of Hair Growth Cycle. It lasts approximately 100 days (3-4 months) and concerns 20% of hair. Hair follicles become inactive and hair is finally shed.
The thinning area of the scalp in which extracted hair follicles arereimplanted during hair transplantation.
A type of reversible hair loss that is mainly noticed in spring and autumn. Occurs due to the seasonal impacts on hair follicles activity.
It lasts approximately 2 months and in case that hair loss persists, further diagnosis should take place.